[Resident Evil] [ABO] Gugela For Private

[-]. Physiological performance of ABO

1. ABO's sexual characteristics

1) Humans have a set of ABO sexual characteristics, and the reproductive organs affected by ABO pheromones are the only reproductive organs of human beings.Although there are differences in physiological structure between men and women, it does not determine the strength of fertility, but only affects the way of sexual behavior and reproductive mode;

3) The pheromones of Alpha and Omega can influence each other, and Alpha and Omega can mark each other, and this mark is mutual.After marking, the marked party is more affected by the marked party's pheromone, and the influence of the unfamiliar pheromone is weakened;

4) There are two ways of marking, which are divided into aging marking and permanent marking. The aging markers between AO are time-sensitive, and the duration of the aging is about three months, which fluctuates depending on the metabolism and contact intensity of both sides of the marker;

5) The permanent marking is also a two-way marking, which requires both AOs who are not in the marking state to mark each other at the same time. time (with a difference of no more than 2 minutes), otherwise an aging mark will be generated.To perform permanent marking again, you need to wait for the aging mark to fade;

6) A very small number of human beings have the androgynous characteristics of AO, BO, and AB at the same time.This feature is mainly manifested in the release of A pheromone outside the estrus period, and the release of O pheromone during the estrus period; no release of pheromone outside the estrus period, and the release of O pheromone during the estrus period;

7) Due to genetic variation, a very small number of humans do not have ABO glands, and such people will always behave as Beta.

2. Physiology and changes of ABO gender

1) Newborn humans have both Alpha and Omega organs, which means that anyone initially has the ability to conceive themselves and make others pregnant, but this organ and the ability to conceive will change with age and AO sex differentiation;

2) The human AO pheromone is released by the gland, and the gland will release the pheromone when it matures, but the pheromone released is affected by both innate and acquired influences, and cannot be confirmed by genetic testing in infancy;

3) AO pheromone affects the maturation and atrophy of human sexual characteristics, but it is not the only influencing factor, and it will also affect the maturity of sexual characteristics the day after tomorrow;

4) A normal human body (even Beta) can metabolize a small amount of A and O pheromones in the body;

5) Human growth is mainly divided into the following stages:

a. The first puberty period (before the age of 18): the sex organ develops, and after the first puberty, it reaches maturity and has fertility, and immature humans do not have fertility;

b. Gland development period (18-20 years old): After becoming fertile, the human body produces gland pheromone, and the gland begins to develop.Immature glands do not release any pheromones.Glands differentiate to release A or O pheromones, or no pheromones as they mature.At this time, although the three types of ABO populations are differentiated in estrus period and estrus mode, their fertility and fertility mode are still completely the same;

c. Metamorphosis period (20-22 years old): For people who do not secrete pheromones, both sets of organs will degenerate, and the activity of sperm and eggs will be greatly reduced; the A pheromone secreted by themselves prevents the degeneration of Alpha□□ organs, Omega□□ organs If it continues to degenerate, it will cause problems of fertilization and implantation difficulties; O pheromone prevents any organ from degenerating.After the degeneration is over, the fertility and fertility patterns of the three groups of ABO populations are differentiated;

d. Stable period (22-50 years old): maintain the state after the end of the metamorphosis period, without changes, except for physiological lesions;

e. Menopause (after the age of 50): The organ degenerates for the second time, and the fertility declines to disappear.This change is for all human beings, regardless of gender and ABO.

3. Gene expression of ABO

1) Today, the following DNA sequences are found on the 23rd pair of human chromosomes, that is, the sex chromosomes, to be related to the development of ABO glands:

a. DXY1001E gene: determines whether humans have ABO gland organs, humans without ABO glands cannot produce ABO pheromones to promote fertility differentiation;

b. DXY1960E gene: determines whether to produce an inhibitory protein that causes the gland to shrink after the gland matures. After the gland shrinks, humans will become Beta because the gland cannot produce A and O pheromones;

c.DXY427E gene: determines whether to produce a catalytic substance that metabolizes A pheromone into O pheromone. In the absence of this substance, the gland releases A pheromone. Otherwise, A pheromone will be converted into O information through a chemical reaction in the gland white.

4. ABO's estrus period

1) Beta has no estrus period; Omega has a estrus period, about once every 3 months, and lasts about 10 days at a time. Omega will not be induced to estrus by other O; Alpha will be induced to estrus when receiving Omega pheromone during estrus;

2) During estrus, Omega will produce a large amount of hormones that promote the production of pheromone metabolites, promote the production of Alpha pheromones, and convert more Alpha pheromones into Omega pheromones.

[-]. ABO modern society

1. Beta humans account for the largest number of humans, accounting for 75% of the total human population, followed by alpha humans, accounting for 15%, and omega humans account for 10%;

2. ABOs are basically equal, but invisible gender discrimination exists, which is similar to the gender equality and discrimination between men and women in the current society;

3. There are a large number of legal and illegal aphrodisiacs, inhibitors, camouflages and other drugs for ABO in the market;

4. Commercially available drugs:

1) Inhibitors: There are corresponding short-acting inhibitors for A and O. Hormones are used to inhibit the release of pheromones. The effect ranges from 1 day to half a month. The effect on O in estrus is weakened. Long-term high-dose use will disrupt the information Normal release of hormones;

2) Aphrodisiac: only for Beta, not effective for AO;

3) Masking agent: Similar to pheromone covering spray, it is safe. Most human A and O use camouflage agent to cover up their own smell and become Beta, but they cannot completely cover up.

5. Black market drugs:

1) Inhibitors: In addition to powerful but more dangerous hormonal drugs, there are also drugs that use targeted drugs to directly block gland growth and pheromone production (such drugs can be replaced by glandectomy). The drug has a great effect on the glands and reproductive organs;

2) Aphrodisiacs: Aphrodisiacs for A and O, forged pheromones or chemically synthesized drugs to induce estrus;

3) Special drugs: medicaments manufactured using various BOW associated products.There are various drugs such as inhibitors, aphrodisiacs, camouflage agents, and even gland repair. Most of them require individual genetic testing and drug development for customers to ensure drug safety, and the prices are high.

[-]. The influence of progenitor virus, LasPlagas parasites and their derivative products on ABO

1. ABO sex characteristics will have different effects on the progenitor virus and its derivative products;

2. ABO sexual characteristics will not affect LasPlagas parasites, but the cross products of parasites and progenitor virus, T virus, G virus and other viruses, such as the second generation of LasPlagas, will be affected;

3. Beta humans mutate into the most common BOW after being infected (such as being infected by T virus to become zombies or infected by C virus to become J'□□o). After infection and mutation, Beta humans will not produce a second mutation (such as become red-eyed zombies or J'□□o secondary mutations or infected with T-Abyss triangular beast drippers, etc.); an exception is the G virus, Beta humans may still mutate after being infected by G, because the G virus is developing A gene that regenerates the gland is inserted into the body, making the infected person appear in a pseudo Alpha state (Omega humans are not affected because they metabolize a pheromone);

4. Alpha humans are prone to secondary or tertiary mutations after infection or parasites to produce secondary or tertiary mutations. The specific mutations are related to the type of virus or parasite.It is more mutable in Alpha, making Alpha a suitable medium;

5. Omega humans are less susceptible to infection because they contain special immunoglobulins. A very small amount of Omega human genes helps to stabilize the virus's traits, so that even after they are infected, the impact can be kept within a controllable range;

6. The BOW produced by this kind of virus whose Omega human genes have stabilized traits can be affected and controlled by specific Omega humans in the early stage (the pheromone receptors are modified to only accept specific pheromones after infection); and the information The source of the hormone and the source of the gene used to stabilize the trait shall be the same person;

7. Since there are very few Omega humans who meet the conditions, this virus only exists in theory at present, and there is no product that can be put into the experiment; the most similar product is the queen bee parasite that appeared in East Slavia in 2012, and this parasite infects the human body Finally, the human body emits specific insect pheromones, which can control the lickers infected by the modified T virus, but the principle is different from the BOW made by using the ABO gene.